Elfishawi, M. M., F. T. Elgengehy, G. I. Mossallam, S. M. Elfishawi, M. M. Alfishawy, and I. M. Metwally,
AB0556 HLA Revisited in Egyptian Patients with Behçet9s Syndrome: New Associations of Hla Alleles with Susceptibility, Protection, Presentation and Severity of The Disaese,
: BMJ Publishing Group Ltd, 2016.
Abstractn/a
Eissa, M., Y. El Miedany, W. Hassan, D. Mekkawy, M. A. Mortada, S. I. Nasef, M. El Gaafary, hala lotfy, yomna farag, G. El Deriny, et al.,
AB0962 UPDATE FOR THE CLINICAL PRACTICE: INTEGRATED, EVIDENCE-BASED APPROACH FOR THE MANAGEMENT OF JUVENILE SPONDYLOARTHRITIS,
: BMJ Publishing Group Ltd, 2019.
Abstractn/a
Eissa, M., Y. El Miedany, W. Hassan, D. Mekkawy, M. A. Mortada, S. I. Nasef, M. El Gaafary, hala lotfy, yomna farag, G. El Deriny, et al.,
AB0962 UPDATE FOR THE CLINICAL PRACTICE: INTEGRATED, EVIDENCE-BASED APPROACH FOR THE MANAGEMENT OF JUVENILE SPONDYLOARTHRITIS,
: BMJ Publishing Group Ltd, 2019.
Abstractn/a
Eissa, M., Y. El Miedany, W. Hassan, D. Mekkawy, M. A. Mortada, S. I. Nasef, M. El Gaafary, hala lotfy, yomna farag, G. El Deriny, et al.,
AB0962 UPDATE FOR THE CLINICAL PRACTICE: INTEGRATED, EVIDENCE-BASED APPROACH FOR THE MANAGEMENT OF JUVENILE SPONDYLOARTHRITIS,
: BMJ Publishing Group Ltd, 2019.
Abstractn/a
Elfishawi, M. M., B. R. Sakr, M. H. El-Arousy, A. K. Hatw, A. N. AbdulKarim, A. B. Tammam, A. N. Kotp, M. E. Hamed, I. E. Genedy, and E. D. El Desouky,
AB1031 Rheumatoid Arthritis, A Single-Center Egyptian Experience,
: BMJ Publishing Group Ltd, 2016.
Abstractn/a
Saeed, D., L. Shalaby, M. Nagy, A. ElZeiny, T. M. Mostafa, M. T. Elrakaiby, M. M. Nooh, A. El-Haddad, M. Abbassi, and R. K. Aziz,
Abstract LB-B19: Investigating the effect of pharmacogenetic variations on voriconazole levels in pediatric cancer patients,
: American Association for Cancer Research, 2019.
Abstractn/a
Kassem, L., S. Omarjee, S. Chabaud, E. Lavergne, C. Faure, F. Beurrier, O. Tredan, L. Corbo, I. Treilleux, and M. Le Romancer,
Abstract P4-11-23: Membranous ERα-36 expression is an independent predictor of poor prognosis in operable breast cancer,
: American Association for Cancer Research, 2015.
Abstractn/a
Kassem, L., L. Fattet, J. Lopez, G. Thibault, E. Lavergne, S. Chabaud, N. Carrabin, N. Chopin, T. Bachelot, and G. Gillet,
Abstract P4-11-24: TIF1 [gamma] interacts with the TGFβ1/SMAD4 signaling leading to poorer outcome in operable breast cancer,
: American Association for Cancer Research, 2015.
Abstractn/a
García-García, R. M., P. G. Rebollar, M. Arias-Álvarez, O. G. Sakr, P. Bermejo-Álvarez, G. Brecchia, A. Gutierrez-Adan, M. Zerani, C. Boiti, and P. L. Lorenzo,
"Acute fasting before conception affects metabolic and endocrine status without impacting follicle and oocyte development and embryo gene expression in the rabbit.",
Reproduction, fertility, and development, vol. 23, issue 6, pp. 759-68, 2011.
AbstractFood deprivation affects female reproduction. The goal of the present study was to elucidate in the rabbit model the effects of acute energy restriction on ovarian function (follicle development, atresia rate and in vitro oocyte maturation) and embryonic development and gene expression of some candidate genes. Serum metabolic parameters (non-esterified fatty acids (NEFA), triglycerides, glucose, insulin and leptin concentrations) and endocrine markers (oestradiol-17β and progesterone concentrations) were also studied. A control group of nulliparous does fed ad libitum and a 72-h fasted group were used. At the end of the nutritional treatment, the ovaries of half of the animals were retrieved while the other animals were re-fed and artificially inseminated to recover embryos at 84 h after insemination, during the luteal phase. At the end of fasting, increased serum NEFA and decreased leptin concentrations were observed in the fasted group, but no differences appeared in serum steroid concentrations, follicle population and atresia rate or nuclear and cytoplasmic oocyte maturation. In the luteal phase, insulin concentrations increased notably in the fasted group. The number of recovered embryos per female and the speed of embryo development were reduced in the food-deprived group. Acute fasting altered both metabolic and endocrine markers and embryo development, but follicle and oocyte development and embryo gene expression were not affected.
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