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Vanacker, H., O. Bally, L. Kassem, and T. Bachelot, "Advanced luminal breast cancer (hormone receptor-positive, HER2 negative): New therapeutic options in 2015", Bulletin du Cancer, vol. 102, issue 6, pp. 47-52, 2015.
Vanacker, H., O. Bally, L. Kassem, O. Tredan, P. Heudel, and T. Bachelot, Advanced luminal breast cancer (hormone receptor-positive, HER2 negative): New therapeutic options in 2015, , vol. 102, issue 6 Suppl 1, pp. S47 - 52, 2015. Abstract
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Vannozzi, A., D. C. J. Wong, J. Höll, I. Hmmam, J. T. Matus, J. Bogs, T. Ziegler, I. Dry, G. Barcaccia, and M. Lucchin, "Combinatorial regulation of stilbene synthase genes by WRKY and MYB transcription factors in grapevine (Vitis vinifera L.)", Plant and Cell Physiology, vol. 59, issue 5, pp. 1043–1059, 2018.
Vannozzi, A., D. C. J. Wong, J. Höll, I. Hmmam, J. T. Matus, J. Bogs, T. Ziegler, I. Dry, G. Barcaccia, and M. Lucchin, "Combinatorial regulation of stilbene synthase genes by WRKY and MYB transcription factors in grapevine (Vitis vinifera L.)", Plant and Cell Physiology, vol. 59, issue 5: Oxford University Press, pp. 1043-1059, 2018. Abstract
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Vannozzi, A., I. Hmmam, J. Holl, J. Bogs, I. Dry, G. Barcaccia, and M. Lucchin, "WRKY transcription factors and regulation of the stilbene biosynthetic pathway in grapevine: new insights and perspectives", Acta Hort, vol. 1188, pp. 1-8, 2017.
Vannozzi, A., I. Hmmam, J. Holl, J. Bogs, I. Dry, G. Barcaccia, and M. Lucchin, "WRKY transcription factors and regulation of the stilbene biosynthetic pathway in grapevine: new insights and perspectives", X International Symposium on Grapevine Physiology and Biotechnology 1188, pp. 1-8, 2016. Abstract
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Vansompel, H., A. Rasekh, A. Hemeida, J. Vierendeels, and P. Sergeant, "Coupled electromagnetic and thermal analysis of an axial flux PM machine", IEEE Transactions on Magnetics, vol. 51, issue 11: IEEE, pp. 1-4, 2015. Abstract
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Vartoukian, S. R., W. M. Tilakaratne, N. Seoudi, M. Bombardieri, L. Bergmeier, A. R. Tappuni, and F. Fortune, "Dysregulation of the suppressor of cytokine signalling 3-signal transducer and activator of transcription-3 pathway in the aetiopathogenesis of Sjögren's syndrome.", Clinical and experimental immunology, vol. 177, issue 3, pp. 618-29, 2014 Sep. Abstract

The suppressor of cytokine signalling 3 (SOCS3) negatively regulates the Janus kinase (JAK)/signal transducer and activator of transcription-3 (STAT-3)/interleukin (IL)-17 pathway. The proinflammatory cytokine IL-17 is over-expressed in Sjögren's syndrome (SS) and is a key factor in its pathogenesis. We hypothesized that IL-17 over-expression in SS results from ineffective regulation by SOCS3. The expression of SOCS3 was analysed in peripheral blood mononuclear cells (PBMC) from SS cases, sicca controls (SC) and healthy controls (HC) and tissue samples from SS, SC and healthy salivary glands (HSG). PBMC and salivary gland tissue from SS and controls were dual-immunostained for SOCS3 and IL-17. IL-6-stimulated PBMC from SS and controls were evaluated for time-dependent STAT-3 activation and SOCS3 induction, and for IL-17 expression. Immunoblotting revealed greater levels of SOCS3 in PBMC from SS than SC (P = 0·017) or HC (P < 0·001). Similarly, the proportion of salivary-gland tissue cells staining for SOCS3 was significantly higher in SS than SC (P = 0·029) or HSG (P = 0·021). The cells in PBMC/salivary gland samples from controls predominantly expressed either SOCS3 or IL-17. However, there was a high frequency of SOCS3/IL-17 co-expression within cells of SS samples. IL-6-stimulation of PBMC from SS cases revealed prolonged activation of STAT-3 with reduced negative regulation by SOCS3, and enhanced expression of IL-17. This study showed that SOCS3 expression is up-regulated in SS. However, the absence in SS of the normal inverse relationship between SOCS3 and pSTAT-3/IL-17 indicates a functional disturbance in this signalling cascade. Consequently, a reduction in function, rather than a reduction in expression of SOCS3 accounts for the unregulated expression of IL-17 in SS, and may play a crucial role in aetiopathogenesis.

Vasa, A. A. K., and et al, "A Female Child with Oligodontia in Primary Dentition – Report of a Rare Case. (Prof. Emad Daif was a peer reviewer for this article).", British Journal of Medicine and Medical Research, vol. 10, issue 12, pp. 1-5, 2015.
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Fayed, A., M. M. El Nokeety, A. A. Heikal, D. O. Abdulazim, M. M. Naguib, and U. A. A. S. E. Din, "Fibroblast growth factor-23 is a strong predictor of insulin resistance among chronic kidney disease patients.", Renal failure, vol. 40, issue 1, pp. 226-230, 2018 Nov. Abstract

Insulin resistance (IR) is very common among chronic kidney disease (CKD) patients. Disturbance in mineral and bone metabolism (MBD) seems to play a role in the pathogenesis of insulin resistance. Fibroblast growth factor-23 (FGF23) is evolving as the most important link between MBD and many pathologic sequences of CKD. The aim was to evaluate IR in pre-dialysis CKD patients looking for a possible association to mineral metabolism among CKD patients. A total of 100 stage 3-5 CKD patients were selected beside 20 normal control subjects. Homeostatic model assessment of insulin resistance (HOMA-IR) was used to assess IR in selected cases. Both groups were compared for fasting blood glucose (FBG), fasting blood insulin (FBI), HOMA-IR, estimated glomerular filtration rate (eGFR), serum calcium (Ca), phosphorus (P), 25 hydroxy vitamin D (25 OH vit D), parathormone (PTH), and uric acid (UA). Correlation study between HOMA_IR and different studied parameters was performed. HOMA-IR is significantly higher in CKD (8.87 ± 3.48 vs. 3.97 ± 0.34 in CKD vs. control, respectively, p < .001). In addition CKD patients have significantly higher FGF23 (235 ± 22.96 vs. 139 ± 12.3 pg/mL, p < .001), PTH (76.9 ± 15.27 vs. 47.9 ± 2.52 pg/mL, p < .001), P (4.3 ± 0.67 vs. 3.6 ± 0.23 mg/dL, p < .001), and UA (5 ± 1.22 vs. 4.85 ± 0.48 mg/dL, p < .001) and significantly lower Ca (8.2 ± 0.3 vs. 8.9 ± 0.33 mg/dL, p < .001), and 25 (OH) vit D (17 ± 5.63 vs. 37 ± 3.43 ng/mL, p < .001). Stepwise linear regression analysis revealed that BMI, GFR, Ca, P, and FGF23 were the only significant predictors of HOMA IR. Increased IR in CKD is a consequence of the uremic status and is intimately associated with disturbed phosphate metabolism and FGF23. Further studies are needed to look for an underlying mechanism.

Fayed, A., M. M. El Nokeety, A. A. Heikal, K. Marzouk, H. Hammad, D. O. Abdulazim, M. M. Salem, and U. A. Sharaf El Din, "Serum 25-hydroxyvitamin D level is negatively associated with serum phosphorus level among stage 3a-5 chronic kidney disease patients.", Nefrologia : publicacion oficial de la Sociedad Espanola Nefrologia, 2018 May 26. Abstract

BACKGROUND: Serum 25-hydroxyvitamin D (25(OH)D) negatively correlates with serum phosphorus level of stage 3a-5 chronic kidney disease (CKD) patients. So far, no explanation has been provided for this negative association.

OBJECTIVE: To confirm this negative association and determine if this relationship is mediated through other known co-morbid factors.

CASES AND METHODS: One hundred (57 male and 43 female) pre-dialysis stage 3a-5 CKD patients were selected. Estimated glomerular filtration rate (eGFR), serum calcium (Ca), phosphorus (P), 25(OH)D, parathyroid hormone (PTH), and intact fibroblast growth factor-23 (FGF23) were assessed. A correlation analysis between serum 25(OH)D and the different parameters studied was performed. Multivariate linear regression analysis was carried out to determine predictors of 25(OH)D.

RESULTS: The negative association between serum 25(OH)D and serum P was confirmed in univariate and multivariate correlation analysis. On the other hand, we failed to detect a significant association between 25(OH)D and serum FGF23. Serum P is the most important independent predictor of 25(OH)D in these patients (partial R=0.15, p<0.0001).

CONCLUSION: Serum P is likely to have a direct negative impact on serum 25(OH)D. Further studies are needed to determine the underlying mechanism.

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Vaselli, M., M. A. Harith, V. Palleschi, G. Salvetti, and D. P. Singh, "Screening effect of impurities in metals: a possible explanation of the process of cold nuclear fusion", Il Nuovo Cimento D, vol. 11, no. 6: Springer, pp. 927–932, 1989. Abstract
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Vassen, W., E. A. J. M. Bente, and W. Hogervorst, "CW laser excitation of the barium 6sng and 6snh Rydberg series from the metastable 6s5d 1.3 D and 5d2 1G4 states", Journal of Physics B: Atomic and Molecular Physics, vol. 20, no. 11: IOP Publishing, pp. 2383, 1987. Abstract
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Vasso Apostolopoulos, Joanna Bojarska, T. - T. C. S. E., R. N. K. Krzysztof Kaczmarek, John Matsoukas, and K. K. E. M. S. I. T. Maha AlKhazindar, Vanessa Barriga, "A global review on short peptides: frontiers and perspectives", Molecules, vol. 26, issue 2, pp. 430-, 2021.
Vater, J., A. S. Bhagwat, M. A. A. M. SHALLAN, A. Blokesch, and J. Salnikow, "DETECTION OF NEAREST-NEIGHBOR RELATIONSHIPS AMONG THE PROTEIN-COMPONENTS OF THE CYTOCHROME B6/F-COMPLEX FROM SPINACH THYLAKOIDS BY CHEMICAL CROSS-LINKING", PHOTOSYNTHESIS RESEARCH, vol. 34, no. 1: KLUWER ACADEMIC PUBL SPUIBOULEVARD 50, PO BOX 17, 3300 AA DORDRECHT, NETHERLANDS, pp. 153–153, 1992. Abstract
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Vavassori, S., J. Chou, L. E. Faletti, V. Haunerdinger, L. Opitz, P. Joset, C. J. Fraser, S. Prader, X. Gao, L. A. Schuch, et al., "Multisystem inflammation and susceptibility to viral infections in human ZNFX1 deficiency.", The Journal of allergy and clinical immunology, 2021. Abstract

BACKGROUND: Recognition of viral nucleic acids is one of the primary triggers for a type I interferon-mediated antiviral immune response. Inborn errors of type I interferon immunity can be associated with increased inflammation and/or increased susceptibility to viral infections as a result of dysbalanced interferon production. NFX1-type zinc finger-containing 1 (ZNFX1) is an interferon-stimulated double-stranded RNA sensor that restricts the replication of RNA viruses in mice. The role of ZNFX1 in the human immune response is not known.

OBJECTIVE: We studied 15 patients from 8 families with an autosomal recessive immunodeficiency characterized by severe infections by both RNA and DNA viruses and virally triggered inflammatory episodes with hemophagocytic lymphohistiocytosis-like disease, early-onset seizures, and renal and lung disease.

METHODS: Whole exome sequencing was performed on 13 patients from 8 families. We investigated the transcriptome, posttranscriptional regulation of interferon-stimulated genes (ISGs) and predisposition to viral infections in primary cells from patients and controls stimulated with synthetic double-stranded nucleic acids.

RESULTS: Deleterious homozygous and compound heterozygous ZNFX1 variants were identified in all 13 patients. Stimulation of patient-derived primary cells with synthetic double-stranded nucleic acids was associated with a deregulated pattern of expression of ISGs and alterations in the half-life of the mRNA of ISGs and also associated with poorer clearance of viral infections by monocytes.

CONCLUSION: ZNFX1 is an important regulator of the response to double-stranded nucleic acids stimuli following viral infections. ZNFX1 deficiency predisposes to severe viral infections and a multisystem inflammatory disease.

Vazhappilly CG, S. E, Ramadan W, Menon V, Al-Azawi AM, Tarazi H, Abdu-Allah H, El-Shorbagi AN, and E. - A. R., "Inhibition of SHP2 by new compounds induces differential effects on RAS/RAF/ERK and PI3K/AKT pathways in different cancer cell types.", Invest New Drugs. 2019 , vol. 37(2), pp. 252-261, 2019. invest_new_drugs_1.pdf
Vázquez, H. C., S. Jäger, W. Wolter, M. Zschöck, M. A. C. Vázquez, and A. Elsayed, Genotyping of Staphylococcus aureus isolated from dairy herds in Mexico, , vol. 21, issue 4: Universidad del Zulia, pp. 308 - 316, 2011. Abstract
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Vázquez, H. C., A. M. I. R. EL SAYED, S. Y. B. I. L. L. E. JAGER, A. L. B. E. R. JOACHIM, C. R. I. S. T. O. P. H. LAMMLER, and W. I. L. F. R. I. E. D. WOTER, Estudio comparativo de las características de Staphylococcus aureus aislados de casos de mastitis clínica y subclínica en México, , vol. 37, issue 002, 2009. Abstract
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Veeramachaneni, C., and et al, "Retreatment efficacy of propoint and guttapercha- A cone beam computed tomography analysis. (Prof. Emad was a peer reviewer for this article).", British Journal of Medicine and Medical Research, In Press.
Veksler, D. B., A. V. Muravjov, Y. V. Kachorovskii, T. A. Elkhatib, K. N. Salama, X. - C. Zhang, and M. S. Shur, "Imaging of field-effect transistors by focused terahertz radiation", Solid-State Electronics, vol. 53, issue 6: Pergamon, pp. 571-573, 2009. Abstract
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