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S, E., B. D, R. H. A. 1, and I. S, "Cosmetic camouflage as an adjuvant to vitiligo therapies: Effect on quality of life", J Cosmet Dermatol ., vol. 20, issue 1, pp. 159-165. , 2021.
S, E. - J., T. HSA, E. MY, G. H, and E. HMS, "Differentiated versus undifferentiated mesenchymal stem cell therapy in paraquat model of Parkinson’s disease", the 20th international congress of Parkinson's disease and movement disorders, Berlin. Germany, 22 June, 2016.
S, E., Hadidi HHE, H. RA, and F. M. M. A. H. D. M. S. O. S. S. O. G. Gawdat HI, Tawdy AM, "Increased tenascin C and DKK1 in vitiligo: possible role of fibroblasts in acral and non-acral disease.", Arch Dermatol Res. ., vol. 310, issue 5, pp. 425-430, 2018.
S, S., E. MF, M. I, E. - S. GS, and R. M, "Aluminium and lead abnormalities in children on haemodialysis: relationship with some medications", Archives of Medical Science, vol. 6, issue 3, pp. 420-429, 2010. aluminium1final_pdf.pdf
S, E., Anbar T, Rasheed H, H. RA, and R. A, "Analysis of symmetricity in the three different (sagittal, transverse and frontal) planes in generalized nonsegmental vitiligo", Indian J Dermatol Venereol Leprol, vol. 87, issue 1, pp. 22-28., 2021.
S, S., R. S, L. HM, E. H. S, M. H, and F. Y, "MEFV gene mutations in Egyptian children with Henoch-Schonlein purpura", Pediatr Rheumatol Online J, issue 12, pp. 14, 2014.
S, E., W. van Geel N, Ezzedine K, Pandya AG, and G. V. B. L. J. E. H. H. J. E. E. L. S. I. A, Bekkenk M, "Validation of a physician global assessment tool for vitiligo extent: Results of an international vitiligo expert meeting.", Pigment Cell Melanoma Res. 2019 Sep;32(5):728-733. doi: 10.1111/pcmr.12784. Epub 2019 May 9., vol. 32, issue 5, pp. 728-733, 2019.
S, M., S. A, C. J, D. C. P, and C. K, "A novel silk suture-assisted laparoscopic technique for the repair of a gastrocolic fistula in a pediatric patient", Journal of Pediatric Surgery, vol. 13, pp. 45-47, 2016.
S, L., S. T, R. M, FarrohKhY, A. Z, and Y. T, "Synthesis, characterization and cytotoxic evaluation of graphene oxide nanosheets: in vitro liver cancer model.", Asian Pac J Cancer Prev , vol. 18, issue 4, pp. 955-961, 2017.
osman. S, A. A. H. Z, A. A. W. E, and undefined, Conflict resolution behaviors among addict adolescents in Egypt, , 2015. artical.pdf
S, A. - A., E. S. A. Gaber O, M. D, A. M, K. EZ, and E. I, " FLT3-ITD Mutations in Egyptian Patients of Acute Myeloid Leukemia: Correlation with Cytogenetic, FAB Subgroups and Prognosis", Clin Lab, vol. May(1)63, issue 5, pp. 1027-1034, 2017.
S, T., G. SM, N. M, N. N, L. D, S. I, and G. TA, "Vascular endothelial growth factor G1612A (rs10434) gene polymorphism and neuropsychiatric manifestations in systemic lupus erythematosus patients.", Rev Bras Reumatol, vol. 57, issue 2, pp. 149-153, 2017. Abstractfinal_accept_published_1-s2.0-s2255502116300980-main.pdfWebsite


To investigate the relation between vascular endothelial growth factor (VEGF) gene polymorphism in systemic lupus erythematosus (SLE) patients and lupus related neuropsychiatric manifestations.

Patients and methods

Sixty adult SLE patients recruited from the Rheumatology and Neurology departments of Cairo University hospitals were classified into two groups; Group A: 30 patients with neuropsychiatric manifestations (NPSLE) and Group B: 30 patients without. For both groups the SNP G1612A (rs10434) of the VEGF gene was genotyped by real time polymerase chain reaction (RT-PCR).


Statistically significant difference was found in genotype and allele frequencies between both groups (AA [70% vs 13.3%, p < 0.001] and GG [10% vs 66.7%, p < 0.001]).


Polymorphism in the gene coding for VEGF may be associated with increased incidence of neuropsychiatric lupus in SLE patients.

S, F., S. Sabet, F. A. Abu Zahra, and A. A. El-Ghor, "Bone marrow derived-mesenchymal stem cells downregulate IL17A dependent IL6/STAT3 signaling pathway in CCl4-induced rat liver fibrosis.", PloS one, vol. 13, issue 10, pp. e0206130, 2018. Abstract

Therapeutic potential of bone marrow-derived mesenchymal stem cells (BM-MSCs) has been reported in several animal models of liver fibrosis. Interleukin (IL) 17A, IL6 and Stat3 have been described to play crucial roles in chronic liver injury. However, the modulatory effect of MSCs on these markers was controversial in different diseases. BM-MSCs might activate the IL6/STAT3 signaling pathway and promote cell invasion in hepatocellular carcinoma, but the immunomodulatory role of BM-MSCs on IL17A/IL6/STAT3 was not fully elucidated in liver fibrosis. In the present study, we evaluated the capacity of the BM-MSCs in the modulation of cytokines milieu and signal transducers, based on unique inflammatory genes Il17a and Il17f and their receptors Il17rc and their effect on the IL6/STAT3 pathway in CCl4-induced liver fibrosis in rats. A single dose of BM-MSCs was administered to the group with induced liver fibrosis, and the genes and proteins of interest were evaluated along six weeks after treatment. Our results showed a significant downregulation of Il17a, Il17ra, il17f and Il17rc genes. In accordance, BM-MSCs administration declined IL17, IL2 and IL6 serum proteins and downregulated IL17A and IL17RA proteins in liver tissue. Interestingly, BM-MSCs downregulated both Stat3 mRNA expression and p-STAT3, while Stat5a gene was downregulated and p-STAT5 protein was elevated. Also P-SMAD3 and TGFβR2 proteins were downregulated in response to BM-MSCs treatment. Collectively, we suggest that BM-MSCs might play an immunomodulatory role in the treatment of liver fibrosis through downregulation of IL17A affecting IL6/STAT3 signaling pathway.

S, I. M., Y. A. Elsayed, H. F. Azzam, and R. E. Reyad, "Effectiveness of diet and exercise program in the clinical features of Polycystic Ovarian Syndrome among adolescents", 4th World Congress on Polycystic Ovarian Syndrome, USA, 26 October , 2018. program.pdf
S, T., F. A, T. S, H. A, Darwish AD, E. T, and D. E., " Increased Serum Endoglin and Transforming Growth Factor β1 mRNA Expression and Risk of Hepatocellular Carcinoma in Cirrhotic Egyptian Patients.", Asian Pac J Cancer Prev. , vol. 17, issue 5, pp. 2429-34, 2016. endoglin_april_2016.pdf