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Web Article
Isaac, S. K., "Awakening Inter-Regionalism? The EU and Regional Arab Organizations Post 2011", GR:EEN Working Paper Series, No. 49, 2014.
Ibrahim, M. A. E., and H. D. Kalfallah, "Effect of Instructions for Mothers Regarding Weaning on Their Infants Complains", IOSR Journal of Nursing and Health Science (IOSR-JNHS) e- ISSN: 2320–1959.p- ISSN: 2320–1940 Volume 7, Issue 3 Ver. III (May-June .2018), PP 31-42 www.iosrjournals.org, Submitted. e0703033142_3.pdf
Isaac, S. K., "The EU's Democracy-Stability Dilemma Persists in Egypt", e-International Relations, 2013.
Isaac, S. K., and H. E. Kares, "The European Community Framing of the Mediterranean 1970-1990", Medreset Working Paper no.1, Rome, Istituto Affari Internazionali (IAI), 2017.
Rashwan, S. S., A. H. Irahim, T. W. Abou-Arab, M. A. Nemitallah, and M. A. Habib, Experimental study of atmospheric partially premixed oxy-combustion flames anchored over a perforated plate burner, , 2017.
Isaac, S. K., "A Quest for Political Stability or Regional Prestige? The Centrality of the Gulf Role in Arab Revolutions Countries", Analysis no. 177, Milan: Istituto per gli Studi di Politica Internazionale , 2013.
Gamal, A. M., A. M. H. Ibrahim, E. S. Mahdi, F. E. Mahallawy, A. Abdelhafez, M. A. Nemitallah, S. S. Rashwan, and M. A. Habib, "Structure and Lean Extinction of Premixed Flames Stabilized on Conductive Perforated Plates", Structure and Lean Extinction of Premixed Flames Stabilized on Conductive Perforated Plates, 2017.
Isaac, S. K., "Understanding the Breadth of the US-Egyptian Discord", Istituto per gli Studi di Politica Internazionale, 2014.
Unpublished
Thesis
Ibrahim, A. M. N. A., 3-Dimentional Ultrasound Imaging For Medical Telesonography: Algorithms and Architectures, , Lausanne, The Ecole polytechnique fédérale de Lausanne ( EPFL), 2018.
ElGhawaby, H. S. H., M. S. A. ElFatah, and M. F. Ibrahim, Accuracy of MDCT in preoperative local staging of colorectal carcinoma, , Cairo, Cairo, 2014.
Ibrahim, F. A. M., Anesthetic considerations in high risk obstetric patients, , Giza, Cairo University, 2009.
Abou-Shady, H., Application of the Eikonal Expansion and Glauber theory to the nuclear reactions, , Cairo- Giza, Cairo University, 1997.
Mohamed, S. N. A., I. M. Ibrahim, and S. M. Younan, Assessment of Cognitive Functions and Some Markers of Synaptic Plasticity in Diabetic Rats, , Cairo, Cairo University, 2012. Abstract

Cognitive dysfunction is a common complication of diabetes mellitus however, less addressed and recognized. This study aimed to investigate the effect of type 1 and 2 diabetes on cognitive functions and related markers of hippocampal synaptic plasticity and the possible impact of blocking N-methyl-d-aspartate (NMDA) receptors by memantine. Seven rat groups were included in this study: non-diabetic, non-diabetic-memantine, type 1 diabetic groups: Untreated, treated with insulin alone and treated with insulin and memantine and type 2 diabetic groups: untreated and memantine treated. Cognitive functions were assessed by Morris Water Maze and passive avoidance test and immunohistochemistry was used for detection of hippocampus pre and post-synaptic markers: synaptophysin and postsynaptic density protein-95(PSD-95) respectively, learning and memory plasticity marker: activity regulated cytoskeletal associated protein (Arc) and the astrocytes reactivity marker: glial fibrillary acidic protein (GFAP). Both type 1 and 2 untreated diabetic groups showed significantly impaired cognitive performance with concomitant decrease in synaptophysin and PSD-95 compared to the non-diabetic group. In addition type 2 group showed a significant decrease in hippocampus GFAP and Arc compared to the non-diabetic group. Treating type 1 diabetic group with insulin alone significantly improved cognitive performance and PSD-95 and significantly decreased GFAP and Arc compared to untreated type 1 group. Blocking NMDA receptors by memantine (30 mg/kg/day) for 3 weeks significantly increased cognitive performance, synaptophysin, GFAP and Arc in type 1 insulin-memantine group compared to type 1-insulin
group and significantly increased synaptophysin, PSD-95 and Arc in type 2-memantine group compared to untreated type 2 diabetic group. In conclusion, cognitive functions are impaired in both types of diabetes mellitus and can be improved by blockage of NMDA receptors which may spark future therapeutic role of these receptors in diabetes-associated cognitive dysfunction.

Mohamed, S. N. A., I. M. Ibrahim, and S. M. Younan, Assessment of Cognitive Functions and Some Markers of Synaptic Plasticity in Diabetic Rats, , Egypt, Cairo University, 2013. Abstractshaimaa_nasr_amin_mohamed.pdf

Cognitive dysfunction is a common complication of diabetes mellitus
however, less addressed and recognized. This study aimed to investigate the
effect of type 1 and 2 diabetes on cognitive functions and related markers of
hippocampal synaptic plasticity and the possible impact of blocking Nmethyl-
d-aspartate (NMDA) receptors by memantine. Seven rat groups
were included in this study: non-diabetic, non-diabetic-memantine, type 1
diabetic groups: Untreated, treated with insulin alone and treated with
insulin and memantine and type 2 diabetic groups: untreated and
memantine treated. Cognitive functions were assessed by Morris Water
Maze and passive avoidance test and immunohistochemistry was used for
detection of hippocampus pre and post-synaptic markers: synaptophysin
and postsynaptic density protein-95(PSD-95) respectively, learning and
memory plasticity marker: activity regulated cytoskeletal associated protein
(Arc) and the astrocytes reactivity marker: glial fibrillary acidic protein
(GFAP). Both type 1 and 2 untreated diabetic groups showed significantly
impaired cognitive performance with concomitant decrease in
synaptophysin and PSD-95 compared to the non-diabetic group. In addition
type 2 group showed a significant decrease in hippocampus GFAP and Arc
compared to the non-diabetic group. Treating type 1 diabetic group with
insulin alone significantly improved cognitive performance and PSD-95
and significantly decreased GFAP and Arc compared to untreated type 1
group. Blocking NMDA receptors by memantine (30 mg/kg/day) for 3
weeks significantly increased cognitive performance, synaptophysin, GFAP
and Arc in type 1 insulin-memantine group compared to type 1-insulin
Best Theses Awards, Cairo University (BTACU)
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group and significantly increased synaptophysin, PSD-95 and Arc in type 2-
memantine group compared to untreated type 2 diabetic group. In conclusion,
cognitive functions are impaired in both types of diabetes mellitus and can be
improved by blockage of NMDA receptors which may spark future therapeutic
role of these receptors in diabetes-associated cognitive dysfunction.

Elawar, A., G. Sayed, N. Ibrahim, and W. Abdelrahman, Assessment of metabolic syndrome in rheumatoid arthritis patients and its realtion to disease activity, , Cairo, Cairo university, In Press.