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Ezzat, M., T. A. ElGhazaly, and M. Gheith, "A Word & Character N-Gram based Arabic OCR Error Simulation model", INTERNATIONAL JOURNAL OF COMPUTERS & TECHNOLOGY, vol. 12, no. 8, pp. 3758–3767, 2014. Abstract
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Samanta, S., D. Kundu, S. Chakraborty, N. Dey, T. Gaber, A. E. Hassanien, and T. - H. Kim, "Wooden Surface Classification based on Haralick and The Neural Networks", Information Science and Industrial Applications (ISI), 2015 Fourth International Conference on: IEEE, pp. 33–39, 2015. Abstract
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Ahmed, A. A., A. A. Mohamed, I. A. Guled, H. M. Elamin, A. H. Abou-Zeid, J. Loko Roka, R. Van den Bergh, S. Au, E. De Plecker, R. Zachariah, et al., Womens/Reproductive Health:[32], , Submitted. Abstract
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Ghani, R. M. A., "Women Believes and Practice toward Cesarean Section Wound Healing", Journal of Biology, Agriculture and Healthcare, vol. 3, 2013. journal_of_biology.pdf
mostafa Goma, H., women and cancer, , 2015.
Abdel-Hameed, A. M., M. E. L. - S. Abuarab, N. Al-Ansari, H. S. Mehawed, M. A. Kassem, H. He, Y. Gyasi-Agyei, and A. Mokhtar, "Winter Potato Water Footprint Response to Climate Change in Egypt", Atةosphere, vol. 13, issue 7, pp. 1-16, 2022.
Samer, M., M. Fiedler, H. - J. Muller, M. Glaser, C. Ammon, W. Berg, P. Sanftleben, and R. Brunsch, "Winter Measurements of Air Exchange Rates Using Tracer Gas Technique And Quantification of Gaseous Emissions From A Naturally Ventilated Dairy Barn", Applied Engineering in Agriculture, 2011. Abstract

Measuring the ventilation rates and then quantifying the gaseous emissions from naturally ventilated barns is a particularly difficult task and associated with large uncertainties; where no accurate, reliable, and online method is available for ventilation rate measurements. Therefore, the objective of this study was to develop further the tracer gas technique (TG) for ventilation rate measurements through winter seasons. Fifteen field experiments were carried out to study the ventilation rates in a naturally ventilated dairy barn located in North Germany through three consecutive winter seasons.

Samer, M., M. Fiedler, H. - J. Müller, M. Gläser, C. Ammon, W. Berg, P. Sanftleben, and R. Brunsch, "Winter measurements of air exchange rates using tracer gas technique and quantification of gaseous emissions from a naturally ventilated dairy barn", Applied engineering in agriculture, vol. 27, issue 6, pp. 1015-1025, 2011.
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Ghaith, W. Z., N. N. Nassar, E. A. Mobarez, and D. M. Abdallah, "THE WIN55, 212-2 ENIGMA IN A RESERPINE INDUCED PARKINSON MODEL", WORLD JOURNAL OF PHARMACY AND PHARMACEUTICAL SCIENCES, vol. 9, issue 7, pp. 2282-2301, 2020.
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Enany, E. G., N. Nagui, H. Nada, S. SHALABY, I. Sany, A. Nada, S. Orabi, E. O. Ghanam, and M. Abdel-Halim, "Widespread Keratotic Spiky Follicular Papules associated With Hyperpigmentation: Challenge", Am J Dermatopathol, vol. 42, issue 10, pp. e134–e135, 2020.
Enany, E. G., N. Nagui, H. Nada, S. SHALABY, I. Sany, A. Nada, S. Orabi, E. O. Ghanam, and A. - M. Halim, "Widespread Keratotic Spiky Follicular Papules Associated With Hyperpigmentation: Answer", Am J Dermatopathol, vol. 42, issue 10, pp. 787–788, 2020.
Eshrah, I. A., A. A. Kishk, A. B. Yakovlev, A. W. Glisson, and C. E. Smith, "Wideband lumped element equivalent circuit model for waveguide slots and applications", Microwave Symposium Digest, 2004 IEEE MTT-S International, vol. 2: IEEE, pp. 607–610, 2004. Abstract
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Abdelwahab, A., J. L. Sapp, M. Gardner, and R. Parkash, "Wide complex tachycardia in a patient with paroxysmal atrial fibrillation", Heart Rhythm, vol. 5, no. 11: Elsevier, pp. 1621–1624, 2008. Abstract
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Abdelwahab, A., J. L. Sapp, M. Gardner, and R. Parkash, "Wide complex tachycardia in a patient with paroxysmal atrial fibrillation", Heart Rhythm, vol. 5, issue 11: Elsevier, pp. 1621-1624, 2008. Abstract
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Abdel-Fattah, M. A. - K., and G. H. Galal-Edeen, "Why the Interpretive Paradigm is Needed for Evaluating e-Government Systems?", Proceedings of the 8th European Conference on Information Warfare and Security, Military Academy, Lisbon and the University of Minho, Braga, Portugal, 6-7 July 2009, Lausanne, Switzerland, pp. 1, 2009. Abstract

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Gregory S. Thomas, L. Samuel Wann, Adel H. Allam, R. C. Thompson, David E. Michalik, M. LindaSutherland, J. D. Suth, I. Guido P. Lombardi, Lucia Watson, Samantha L. Cox, et al., "Why did ancient people have atherosclerosis?: From autopsies to computed tomography to potential causes", GLOBAL HEART, vol. 9, issue 2, 2014.
Abdel-Fattah, M. A. - K., and G. H. Galal-Edeen, "Why an interpretive paradigm is needed for evaluating e-government systems", 9th European Conference on e-Government, pp. 1–10, 2009. Abstract
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Hawary, E. R., S. Meshaal, A. A. Mauracher, L. Opitz, A. D. Elaziz, S. Lotfy, A. Eldash, J. Boutros, N. Galal, P. J. Schmid, et al., "Whole‐exome sequencing of T‐B+ severe combined immunodeficiency in Egyptian infants, JAK3 predominance and novel variants", Clinical and experimental immunology, vol. 203, issue 3, pp. 448-457, 2021.
Etemadnia, H., A. H. M. E. D. HASSAN, S. Goetz, and K. Abdelghany, "Wholesale Hub Locations in Food Supply Chains", Transportation Research Record, vol. 2379, no. 1, pp. 80-89, 2013. AbstractWebsite

This paper addresses the wholesale hub location problem in food supply chains. The paper aims to design an optimal hub location network to serve food consumption markets through efficient connections with production sites. These optimal locations can be compared with the current locations of hubs to determine whether changes could lead to greater efficiencies. The model is mathematically formulated as a mixed-integer programming problem. The model minimizes the total network costs, which include the transportation of goods and the construction of hubs. The mathematical program considers several constraints on travel distance, hub capital cost and capacity, road condition, and transportation cost. Several experiments are conducted to test the sensitivity of the model to changes in parameters such as the food's average travel distance, the maximum hub capacity, and the transportation cost. Then, a real-world application is made to the Northeast United States livestock industry. Finally, the results show the effect of the changes in model parameters on the optimal hub network design (i.e., the number of hubs and the selection of hub locations).

FH Lv, YH Cao, GJ Liu, LY Luo,......., H.E. Rushdi,......., and M. H. Li, "Whole-Genome Resequencing of Worldwide Wild and Domestic Sheep Elucidates Genetic Diversity, Introgression, and Agronomically Important Loci", Molecular biology and evolution, vol. 39, issue 2, pp. 1-26, 2022.
Warejko, J. K., W. Tan, A. Daga, D. Schapiro, J. A. Lawson, S. Shril, S. Lovric, S. Ashraf, J. Rao, T. Hermle, et al., "Whole Exome Sequencing of Patients with Steroid-Resistant Nephrotic Syndrome.", Clinical journal of the American Society of Nephrology : CJASN, vol. 13, issue 1, pp. 53-62, 2018 Jan 06. Abstract

BACKGROUND AND OBJECTIVES: Steroid-resistant nephrotic syndrome overwhelmingly progresses to ESRD. More than 30 monogenic genes have been identified to cause steroid-resistant nephrotic syndrome. We previously detected causative mutations using targeted panel sequencing in 30% of patients with steroid-resistant nephrotic syndrome. Panel sequencing has a number of limitations when compared with whole exome sequencing. We employed whole exome sequencing to detect monogenic causes of steroid-resistant nephrotic syndrome in an international cohort of 300 families.

DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: Three hundred thirty-five individuals with steroid-resistant nephrotic syndrome from 300 families were recruited from April of 1998 to June of 2016. Age of onset was restricted to <25 years of age. Exome data were evaluated for 33 known monogenic steroid-resistant nephrotic syndrome genes.

RESULTS: In 74 of 300 families (25%), we identified a causative mutation in one of 20 genes known to cause steroid-resistant nephrotic syndrome. In 11 families (3.7%), we detected a mutation in a gene that causes a phenocopy of steroid-resistant nephrotic syndrome. This is consistent with our previously published identification of mutations using a panel approach. We detected a causative mutation in a known steroid-resistant nephrotic syndrome gene in 38% of consanguineous families and in 13% of nonconsanguineous families, and 48% of children with congenital nephrotic syndrome. A total of 68 different mutations were detected in 20 of 33 steroid-resistant nephrotic syndrome genes. Fifteen of these mutations were novel.,,, andwere the most common genes in which we detected a mutation. In another 28% of families, we detected mutations in one or more candidate genes for steroid-resistant nephrotic syndrome.

CONCLUSIONS: Whole exome sequencing is a sensitive approach toward diagnosis of monogenic causes of steroid-resistant nephrotic syndrome. A molecular genetic diagnosis of steroid-resistant nephrotic syndrome may have important consequences for the management of treatment and kidney transplantation in steroid-resistant nephrotic syndrome.

Braun, D. A., M. Schueler, J. Halbritter, H. Y. Gee, J. D. Porath, J. A. Lawson, R. Airik, S. Shril, S. J. Allen, D. Stein, et al., "Whole exome sequencing identifies causative mutations in the majority of consanguineous or familial cases with childhood-onset increased renal echogenicity.", Kidney international, 2015 Oct 21. AbstractWebsite

Chronically increased echogenicity on renal ultrasound is a sensitive early finding of chronic kidney disease that can be detected before manifestation of other symptoms. Increased echogenicity, however, is not specific for a certain etiology of chronic kidney disease. Here, we performed whole exome sequencing in 79 consanguineous or familial cases of suspected nephronophthisis in order to determine the underlying molecular disease cause. In 50 cases, there was a causative mutation in a known monogenic disease gene. In 32 of these cases whole exome sequencing confirmed the diagnosis of a nephronophthisis-related ciliopathy. In 8 cases it revealed the diagnosis of a renal tubulopathy. The remaining 10 cases were identified as Alport syndrome (4), autosomal-recessive polycystic kidney disease (2), congenital anomalies of the kidney and urinary tract (3), and APECED syndrome (1). In 5 families, in whom mutations in known monogenic genes were excluded, we applied homozygosity mapping for variant filtering and identified 5 novel candidate genes (RBM48, FAM186B, PIAS1, INCENP, and RCOR1) for renal ciliopathies. Thus, whole exome sequencing allows the detection of the causative mutation in 2/3 of affected individuals, thereby presenting the etiologic diagnosis, and allows identification of novel candidate genes.Kidney International advance online publication, 21 October 2015; doi:10.1038/ki.2015.317.

Daga, A., A. J. Majmundar, D. A. Braun, H. Y. Gee, J. A. Lawson, S. Shril, T. Jobst-Schwan, A. Vivante, D. Schapiro, W. Tan, et al., "Whole exome sequencing frequently detects a monogenic cause in early onset nephrolithiasis and nephrocalcinosis.", Kidney international, vol. 93, issue 1, pp. 204-213, 2018 Jan. Abstract

The incidence of nephrolithiasis continues to rise. Previously, we showed that a monogenic cause could be detected in 11.4% of individuals with adult-onset nephrolithiasis or nephrocalcinosis and in 16.7-20.8% of individuals with onset before 18 years of age, using gene panel sequencing of 30 genes known to cause nephrolithiasis/nephrocalcinosis. To overcome the limitations of panel sequencing, we utilized whole exome sequencing in 51 families, who presented before age 25 years with at least one renal stone or with a renal ultrasound finding of nephrocalcinosis to identify the underlying molecular genetic cause of disease. In 15 of 51 families, we detected a monogenic causative mutation by whole exome sequencing. A mutation in seven recessive genes (AGXT, ATP6V1B1, CLDN16, CLDN19, GRHPR, SLC3A1, SLC12A1), in one dominant gene (SLC9A3R1), and in one gene (SLC34A1) with both recessive and dominant inheritance was detected. Seven of the 19 different mutations were not previously described as disease-causing. In one family, a causative mutation in one of 117 genes that may represent phenocopies of nephrolithiasis-causing genes was detected. In nine of 15 families, the genetic diagnosis may have specific implications for stone management and prevention. Several factors that correlated with the higher detection rate in our cohort were younger age at onset of nephrolithiasis/nephrocalcinosis, presence of multiple affected members in a family, and presence of consanguinity. Thus, we established whole exome sequencing as an efficient approach toward a molecular genetic diagnosis in individuals with nephrolithiasis/nephrocalcinosis who manifest before age 25 years.

Winkler, A. S., M. Leonardi, B. D. Michael, F. Abd-Allah, W. Carroll, and A. Guekht, "A WHO resolution on epilepsy and other neurological disorders.", The Lancet. Neurology, vol. 20, issue 3, pp. 171-172, 2021.
Ghanem, G. A. M., and K. S. Abdulaziz, M. Al-Ajlan and Abdulsalam, "A whitefly-transmitted geminivirus infecting bean ( Phaseolus vulgaris L.) plants in Saudi Arabia.", Egypt . J. Phytopathol, vol. 31, issue 1-2, pp. 1-15, 2003. Abstract
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