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El-Darouti, M. A., S. A. Marzouk, M. R. E. Abdel-Halim, A. Z. Zidan, and M. M. Fawzy, "Basaloid follicular hamartoma", International journal of dermatology, vol. 44, no. 5: Wiley Online Library, pp. 361–365, 2005. Abstract
El-Darouti, M. A., S. A. Marzouk, M. Bosseila, O. abu Zeid, O. El-Safouri, A. Zayed, A. El-Ramly, and M. R. E. Abdel-Halim, "Microscopic study of normal skin in cases of mycosis fungoides", International journal of dermatology, vol. 45, no. 9: Wiley Online Library, pp. 1043–1046, 2006. Abstract
El-Darouti, M. A., S. A. Marzouk, O. Azzam, M. M. Fawzi, M. R. E. Abdel-Halim, A. A. Zayed, and T. M. Leheta, "Vitiligo vs. hypopigmented mycosis fungoides (histopathological and immunohistochemical study, univariate analysis)", European Journal of Dermatology, vol. 16, no. 1, pp. 17–22, 2006. Abstract
El-Darouti, M. A., H. M. Mashaly, E. El-Nabarawy, A. M. Eissa, M. R. E. Abdel-Halim, M. M. T. Fawzi, N. H. El-Eishi, S. O. Tawfik, N. S. Zaki, A. Z. Zidan, et al., "Leukocytoclastic vasculitis and necrolytic acral erythema in patients with hepatitis C infection: Do viral load and viral genotype play a role?", Journal of the American Academy of Dermatology, vol. 63, no. 2: Elsevier, pp. 259–265, 2010. Abstract
El-Mofty, M., M. El-Darouti, H. RASHEED, D. A. Bassiouny, M. Abdel-Halim, N. S. Zaki, G. El-Hanafy, H. El-Hadidi, O. Azzam, A. El-Ramly, et al., "Sulfasalazine and pentoxifylline in psoriasis: a possible safe alternative", Journal of Dermatological Treatment, vol. 22, no. 1: Informa Healthcare USA on behalf of Informa UK Ltd. London, pp. 31–37, 2011. Abstract
Zayed, A. A., M. R. E. Abdel-Halim, K. S. Sayed, F. N. Mohammed, D. M. Hany, and K. S. Amr, "Transforming growth factor-$\beta$1 gene polymorphism in mycosis fungoides", Clinical and experimental dermatology, vol. 39, pp. 806–809, 2014. Abstract
Zayed, A., M. Abdel-Halim, K. SAYED, and K. A. Faisal Nouredin, Doaa Hany, "Gene expression of FOXP3 and TGF-β1 in the blood of patients with mycosis fungoides, a hospital-based case–control study", EWDS, vol. 13, issue 3, pp. 133-136, 2016.
Youssef, R., D. Mahgoub, O. A. Zeid, D. M. Abdel-Halim, M. El-Hawary, M. F. Hussein, M. A. Morcos, D. M. Aboelfadl, H. A. Abdelkader, Y. Abdel-Galeil, et al., "Hypopigmented Interface T-Cell Dyscrasia and Hypopigmented Mycosis Fungoides: A Comparative Study.", The American Journal of dermatopathology, vol. 40, issue 10, pp. 727-735, 2018 Oct. Abstract

Hypopigmented interface T-cell dyscrasia (HITCD) is a distinct form of lymphoid dyscrasia that may progress to hypopigmented mycosis fungoides (HMF). We compared both diseases as regards their CD4/CD8 phenotype and expression of granzyme B and tumor necrosis factor-alpha (TNF-α) and how these are affected by narrow-band UVB (nb-UVB). The study included 11 patients with HITCD and 9 patients with HMF. They received nb-UVB thrice weekly until complete repigmentation or a maximum of 48 sessions. Pretreatment and posttreatment biopsies were stained using anti CD4, CD8, TNF-α, and granzyme B monoclonal antibodies. Epidermal lymphocytes were CD8 predominant in 54.5% and 66.7% of HITCD and HMF cases, respectively, whereas dermal lymphocytes were CD4 predominant in 63.6% and 66.7%, respectively. Significantly, more dermal infiltrate was encountered in HMF (P = 0.041). In both diseases, granzyme B was only expressed in the dermis, whereas TNF-α was expressed both in the epidermis and dermis. No difference existed as regards the number of sessions needed to achieve repigmentation or cumulative nb-UVB dose reached at end of study. (P > 0.05). Narrow-band UVB significantly reduced only the epidermal lymphocytes in both diseases (P ≤ 0.05) with their complete disappearance in 8 (72.7%) HITCD and 6 (66.7%) HMF cases. In both diseases, nb-UVB did not affect granzyme B or TNF-α expression (P > 0.05). In conclusion, both diseases share the same phenotype, with HITCD being a milder form of T-cell dysfunction. In both diseases, epidermal lymphocytes are mainly CD8-exhausted cells lacking cytotoxicity, whereas dermal cells are mostly reactive cells exerting antitumor cytotoxicity. Tumor necrosis factor-alpha mediates hypopigmentation in both diseases and prevents disease progression. Repigmentation after nb-UVB in both diseases occurs before and independently from disappearance of the dermal infiltrate.